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HIV testing?


Cal Crilly

Is it HIV testing?
Robert Gallo was a cancer virus specialist so when he found gay men suffering cancers from lifestyle and lubricant contamination back in the 1980's he found retroviruses only because the gay men they had gathered from different locations in the USA had cancers.
If anyone tests to HIV they can have cancers or autoimmune disease or simply be a pregnant woman.
Anyone who has tested positive after tranfusion could have cancer, an autoimmune disease, be pregnant or at the wrong time of the month when estrogen is high as estrogen encourages retroviruses along with vitamin A.
It is quite serious because anyone who believes they were infected with transfusion will take AIDS drugs.

Anyone who believes they were infected sexually will take AIDS drugs, all for no reason.

You even find in one of Gallo's early studies this comment.

"The amount of complex-bound p24 in sequential serum samples correlated roughly with tumour cell mass."
So p24 goes up with cell growth either in cancers, reproductive tissue or fetuses.
They dilute the HIV antibody test 400 times because otherwise everyone is HIV positive.

P24 turns up in breast tumours too.

And even the former head of the Swiss Blood bank Alfred Hassig said HIV antibodies are actin antibodies and cardiolipin antibodies.
It gets far more complex and he is never quoted but the HIV antibody test is not for a retrovirus.
I've listed the HIV antigens that appear in tumours and more below.

"The major antibody specificities detected in HIV-1 WB analysis include gp160, gp120, p65, p55, gp41, p40, p31, and p24. To be reported as positive, the WB assay requires reactivity against the gp41 and gp120/160 bands encoded by the env (gp160, envelope glycoprotein) gene or against either one of these env bands plus the p24 band encoded by gag [Pr55(Gag)]."
Interpretation of HIV Serologic Testing Results

p24 also turns up in cell trafficking.

"The trafficking of proteins in the secretory pathway is mediated by vesicles. Proteins of the p24 family are present on the membranes of secretory pathway organelles (ER, Golgi, COPI and COPII vesicles). Evidence exists showing that p24 proteins play a role in the development of Alzheimer's disease, making them an interesting research subject. Their presence in the secretory pathway and their tissue-dependent expression levels suggest that p24 proteins are involved in secretion. However, their molecular function is not clear. Several potential functions have been proposed for p24 proteins: (1) that they function as receptors for selected cargo; (2) that they regulate vesicle biogenesis; (3) that they perform structural and morphogenetic functions in the secretory pathway; (4) that they are responsible for quality control of transported proteins. In this article, we provide a critical review of the postulated functions of p24 proteins." 2010
The p24 family proteins--regulators of vesicular trafficking

We find cell transport with gp120 from 'HIV' in a glucose transport protein called GLUT4.

"Our previous studies demonstrated that the expression of gp160 is also limited to fat and muscle tissues, where it is localized exclusively in GLUT4-containing vesicles, and thus, it represents a marker protein for insulin-activated glucose transport." 1994
The major protein of GLUT4-containing vesicles, gp160, has aminopeptidase activity

We find gp120 and p24 in the placentas of women who were not HIV positive.

"Antigens gp120 and p17 were identified in normal chorionic villi in vimentin-positive fibroblast-like cells and in endothelium, respectively. Antigen p24 was localized to HLA-DR positive cells that morphologically resembled macrophages in areas of villitis." 1991
HIV proteins in normal human placentae.

And gp160 in normal placentas.

"Expression of intact endogenous retroviruses by normal placental villous trophoblast and immuno-crossreactivity of villous trophoblast with anti-retroviral antisera have been documented. The nature and/or potential function of these particles/proteins has not yet been fully defined. We previously reported that monoclonal antibodies directed against HIV-1 envelope and gag proteins react with normal human villous trophoblast. In this study, we report that extravillous trophoblast (EVT) from second- and third-trimester tissue are also cross-reactive with anti-HIV-1 gp120/160 and p17/18 antibodies." 1995
Expression of endogenous HIV-1 crossreactive antigens within normal human extravillous trophoblast cells.

And gp120 and gp41 in tumours of reproductive tissue.

"RAK antigens p120, p42, and p25 exhibit molecular and immunological similarity to the proteins encoded by human immunodeficiency virus type 1 (HIV-1) and are expressed by 95% of breast and gynecological cancer cases in women and prostate cancer cases in men.

"Breast cancer DNAs of 40 patients were PCR amplified with HIV-1 gp41-derived primers, and all of the samples were found to be positive. The DNA fragments amplified in seven blindly selected breast cancer samples were sequenced. The breast cancer DNA sequences showed at least 90% homology to the HIV-1 gene for gp41. Antisense oligonucleotides complementary to the HIV-1-like sequences inhibited reverse transcriptase activity and inhibited the growth of breast cancer cells in vitro. Viral particles detected in breast cancer cell lines were strongly immunogold labeled with the anti-HIV-1 gp120 MAb. The results obtained strongly suggest that the long-postulated breast cancer virus may, in fact, be related to HIV-1." 1998
Human Immunodeficiency Virus Type 1-Like DNA Sequences and Immunoreactive Viral Particles with Unique Association with Breast Cancer

In ovarian cancers with gp41.

"PCR with HIV-1 Env-derived primers revealed DNA sequences with over 90% homology to HIV-1 gp41 in syncytia and in ovarian cancer cells but not in normal ovary cells." 1999
Giant Syncytia and Virus-Like Particles in Ovarian Carcinoma Cells Isolated from Ascites Fluid

And cervical cancers with both p24 and gp41.
The gag and env glycoproteins are found using the PCR test so again this test is not valid for HIV but the sequences appear in tumours.

"While investigating whether proteins retrieved by cervicovaginal lavages (CVL) from women with cervical intraepithelial neoplasia (CIN) might correlate with risk of progression to invasive cervical cancer, we unexpectedly identified HIV gag and env glycoprotein in CVL from women with HIV-negative serology. HIV antigens were consistently identified by mass spectrometry (MS) in CVL from 4 women but were absent in CVL from the remaining 16 women. HIV serologies of all 20 patients were negative for both HIV-1 and HIV-2 antibodies."
"WB and IC results demonstrated the presence of HIV-1 gp41 and p24 antigens in four CVL that were identified by mass spectrometry to have the HIV proteins. Despite negative serology, HIV RNA in CVL and HIV p24 in cervix biopsies were detected in patients with HIV antigen-positive CVL. HIV p24-positive CVL were PSA negative.
All 20 subjects remained HIV seronegative throughout the study. Women with HIV proteins and RNA were comparatively older." 2009
Human immunodeficiency virus (HIV) antigens and RNA in HIV-seronegative women with cervical intraepithelial neoplasia

With gp41 in prostrate cancer too.

"Breast and gynecological cancer-associated antigens RAK p120, p42 and p25 exhibit molecular, immunological and genetic homology to HIV-1 proteins. Normal tissues, including the majority of tissues adjacent to cancer, do not express these unique cancer markers. Antigens RAK are now detected in 100% of prostate cancer and in the majority of prostate benign hyperplasia (BPH) cases. Polymerase chain reaction (PCR) with HIV-1 gp41-derived primers revealed prostate cancer-associated DNA fragments of similar size (140 bp) as in HIV-1 genome. Ninety-five percent of BPH samples obtained from prostate cancer patients tested PCR-positive. For comparison, only 61.9% of BPH samples obtained from cancer-free patients tested PCR-positive. The DNA fragments amplified in prostate cancer and in BPH showed more than 90% homology to the HIV-1 gene for gp41. The obtained results strongly suggest that a retrovirus related to HIV-1 may be associated with cancers of the reproductive system." 1998
Prostate, breast and gynecological cancer markers RAK with homology to HIV-1

What they fail to mention is that retroviruses are always in male reproductive tissues like testis.

"The expression of ERVs in male reproductive tissues suggests a possible role in normal and disease conditions involving the testis and epididymis. These speculative functions may include among others spermatogenesis and or sperm maturation or tumour formation. However, further studies need to be carried out to investigate specific roles of ERVs in male reproductive events." 2002
Endogenous retrovirus sequences expressed in male mammalian reproductive tissues: a review.

And retroviruses always appear in the placenta, breasts and cervix of women.

"Retrovirus-like sequences account for 8 to 9% of the human genome."

"In the present study, we have investigated the transcriptional activity of representative members of 20 HERV families in 19 different normal human tissues. Qualitative evaluation of chip hybridization signals and quantitative analysis by real-time RT-PCR revealed distinct HERV activity in the human tissues under investigation, suggesting that HERV elements are active in human cells in a tissue-specific manner. Most active members of HERV families were found in mRNA prepared from skin, thyroid gland, placenta, and tissues of reproductive organs. In contrast, only few active HERVs were detectable in muscle cells."
Comprehensive Analysis of Human Endogenous Retrovirus Transcriptional Activity in Human Tissues with a Retrovirus-Specific Microarray

So the HIV test is not valid or specific to 'HIV' and should only be used to screen blood with possible retroviruses from tumours and people with autoimmune disease but they won't kill you.
It was also made as a blood screening test and was never meant to be used on people.
This is why all the test kits say "this is not to be used for a HIV diagnosis"
In court it should be a simple commercial fraud case to take it off the market.
These are problems with the HIV antibody test in their own words so you can decide.
Most of these problems arise from inflammation, too much cell growth and antibodies to HLA genes.
Many disease conditions can cause hypomethylation of the genome and this makes our retroviruses come out of our DNA.
But none of this makes HIV 'the specific' retrovirus involved.
The retroviruses are always there and the antibodies are not to retroviruses anyway.

'False-positive HIV serologic screens can be caused by recent influenza vaccination, incidental viral infections, autoimmune disease, renal failure, cystic fibrosis, multiple pregnancies, blood transfusions, liver diseases, parenteral substance abuse, hemodialysis, or vaccinations against hepatitis B and rabies.

- An indeterminate WB result can be caused by a weak titer of anti-HIV-1 antibodies (as seen in early seroconversion), advanced AIDS, infection with an unusual HIV type, or recipients of experimental HIV vaccines. It can also be caused by the presence of antibodies cross-reactive against HIV antigens (incidental viral infection; vaccination against influenza, hepatitis, or rabies; or HTLV infection) or reactivity to the nonviral components of the WB (various autoimmune disorders, multiple pregnancies, and recipients of multiple blood transfusions).

- An increasing number of recipients of experimental HIV vaccines, which can cause false-positive results in HIV serologic tests, are being offered HIV screening. Whenever possible, testing for HIV in such patients is best performed in consultation with the vaccine research group responsible for the trial. This procedure will ensure proper interpretation of test results without compromising the study data.'

Interpretation of HIV Serologic Testing Results

Helical Sun


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