In particular, the C677T polymorphism shows a wide regional and ethnic variation. Homozygosity (TT) among Whites is 6-14%. In African populations and in Blacks living outside of Africa such as in Brazil and in the United States, the frequency falls to less than 2% for the TT variant. The prevalence rises in Mediterranean and Hispanic population. For example, among Hispanics in prevalence ranges as high as 21%. Northern China and Japan show an 18 and 12% prevalence respectively" MTHFR Mutation: A Missing Piece in the Chronic Disease Puzzle
Many Hispanics, Caucasians and Jewish Ashkenazis can be missing the folate processing genes MTHFR1 and C677T, in general Westerners eat enough good food and don't get low levels of folate leading to health problems but if you take party drugs or any doctor drugs, the liver can't break down the drugs due to the lower folate and you get nutrient loss which leads to genome hypomethylation and this makes the genes switch on and retroviruses appear in our our DNA. Africans don't have the folate gene loss but they have blood differences in HLA antigens and Sickle Cell, the iron overload can also cause inflammation and retroviral expression.
So people missing the folate processing gene MTHFR get decreased methylation if they don't eat enough folate, simply drinking alcohol can bring on the retroviruses, any exposure to other drugs can as well.
Inflammation is part and parcel of new cell growth because the cell damage causes inflammation and new growth, everything that is anti-inflammatory is anti-HIV and also an antiviral in many cases.
The catch 22 is that CD4 T-cells cause the very same inflammation which causes cell growth and retroviruses to appear.
To say the entity HIV to be the cause of T-cell death is adding 2 + 2 into the wrong sum.
The retroviruses are appearing with new cell growth because the T-cells have gathered at a site of cell inflammation and are sending out signals via cytokines to other immune cells to clear up the mess and fix the damage with new cells.
It's all jumbled together, damage, cells dying, being removed, new cells plugging the gaps.
All of HIV/AIDS is about structure of the body and changes we don't understand yet but we treat our own living tissue as pathogens.
And we poison ourselves.
Here is a study on Jewish people missing MTHFR1 genes, when they say "this reaction" they are talking about a step in methylation and the lack of a Folate gene lowers S-adenosylmethionine, the comment about lipid methylation is also important because the lack of methylation in lipids means the body can react to oxidised fats and create the sort of antibodies that cross react with the HIV antibody test.
"An increase in homocysteine" is accompanied by inflammation.
"This reaction is important for the synthesis of S-adenosylmethionine (SAM), the major intracellular methyl donor for DNA, protein, and lipid methylation reactions [2,3]. Reduced MTHFR activity results in an increased requirement for folic acid to maintain normal homocysteine remethylation to methionine. In the absence of sufficient folic acid, intracellular homocysteine accumulates, methionine resynthesis is reduced, and essential methylation reactions are compromised. An increase in homocysteine and a decrease in methionine result in a decreased ratio of SAM to S-adenosylhomocysteine (SAH), which has been associated with DNA hypomethylation"
"Reduced MTHFR activity (in the Jewish subjects) results in an increased requirement for folic acid ( they need more folate) to maintain normal homocysteine remethylation to methionine. In the absence of sufficient folic acid, intracellular homocysteine accumulates (causing inflammation), methionine resynthesis is reduced, and essential methylation reactions are compromised."
Essential methylation reactions are needed to keep the retroviruses dormant.
HIV is an example.
"However, the role of DNA methylation in the control of HIV-1 latency has never been unambiguously demonstrated, in contrast to the apparent importance of transcriptional interference and chromatin structure, and has never been studied in HIV-1-infected patients."
"In the latent reservoir of HIV-1-infected individuals without detectable plasma viremia, we found HIV-1 promoters and enhancers to be hypermethylated (the methylation kept them dormant) and resistant to reactivation, as opposed to the hypomethylated 5? LTR in viremic patients." (the hypomethylation gave them a viral load or viremia)
"the role of DNA methylation has never been studied in HIV-1-infected patients" that was a 2009 study so we are only on the cusp of even thinking about using the methylation nutrients Selenium, Folate, B12, B6, Choline to make S-Adenosylmethionine and prevent genome hypomethylation to control our retroviruses.
Folate and B12 loss affects platelet formation in hemophilia and are the main anemia co-factors too so you see any lack of these methylation nutrients can make our retroviruses express, with either anemia or iron overload.
Hemophiliacs may unfortunately be expressing retroviruses anyway due to low Folate and B12.
And many hemophiliacs may simply be missing these folate genes.
The HIV/AIDS 'misunderstanding' began because Richard Nixon gave people like Gallo lots of money in his famous "War on Cancer" which went nowhere.
Robert Gallo found his cancer viruses in the form of retroviruses but they didn't always infect other cells or tissue.
If cells or tissue cultures are hypomethylating the retroviruses can jump into the DNA but this is not an infection.
Retroviruses are not pathogens thinking to themselves "Oh I'll jump on this sperm and travel from Africa to Haiti onto New York and San Francisco and I'll find some gays to infect".
All of this is imaginary bias on the behalf of scientists.
8% of our DNA is retrovirus on the last count.
They add in numbers like 40 million infected with AIDS retroviruses in Sub-Saharan Africa because that's their own racist bias, the numbers do not add up, people are already crawling with retroviruses, this is all cashing in on fear of the unknown.
All of the HIV antigens appear in most diseases and are in all sorts of animals, fungi etc.
There were 2 waves of poisoning in the gays, the first was because a giant party with lots of drugs had happened in the late 70's and because the gay ghettoes were surrounded by the Reagan government's America any party liver damage or infections were treated with seriously dangerous experimental medication even before AIDS happened or AZT was allowed.
The gay community were also sold Hot Oil sexual lubricants with a level of Benzene that the FDA quietly lowered years after the epidemic of poisoning had got into full swing.
The poppers gays use as sexual stimulants cause thymus atrophy and hence no T-cells can be made, cocaine does the same.
Benzene is still in the sexual lubricants but at lower levels.
The second wave was caused by AZT, the dose from 1987 to 1990 was high dose AZT monotherapy at 1000-1200mg a day.
Almost everyone died on it.
What they did was jumble a few new chemicals together to stop reverse transcriptase and cell growth and called it HAART.
The essential difference is the AZT dose has been lowered so people now live longer and but still get poisoned slowly.
They are talking about lowering the dose of AZT further.
Whereas iron status in AIDS patients is the clear predictor of whether they will live or die.
Step back and the entire thing is a train crash.
And some people missing a few genes may have got caught in the wreckage.